Question Type:
Strengthen
Stimulus Breakdown:
Conclusion: The bacteria's exposure to heavy metals in the sludge has promoted their resistance to antibiotics.
Evidence: Bacteria that survive in heavy-metal-filled sludge have evolved unusual ability to resist heavy-metal poisoning, and they show a strong resistance to antibiotics.
Answer Anticipation:
In abstract terms, this author seems to think that since X (bacteria) now shows trait A (resistance to heavy-metal poisoning) and trait B (resistance to antibiotics), it's probably the case that trait A caused trait B. This is quite dubious. Since the conclusion is causal, we should go to our two causal pressure points:
1. Is there some other way to explain the same evidence?
2. What additional info could impact the plausibility of the author's explanation?
For #1, we could say trait A and B are simply coincidental. Neither causes the other. We might also say that trait B causes trait A (resistance to antibiotics is what provides resistance to heavy-metal poisoning). Or perhaps it's simply some other factor, for example the ammonia in sewage sludge, that transforms the bacteria into being both trait A and B. Correct strengthen answers can simply rule out an alternative explanations.
#2 answers could sound like Covariation (cause/effect or no-cause/no-effect). It could sound like "bacteria in sludge that DOESN'T contain heavy metals, does NOT develop resistance to antibiotics". It could simply verify that supposed-cause comes before supposed-effect, "The bacteria did NOT have a resistance to antibiotics UNTIL it was subjected to heavy metal exposure". It could also provide other examples of this connection between heavy metals and antibiotic resistance, or it could explain the causal mechanism of how this would work, i.e. "the heavy metals ionize some of the ingredients in antibiotics, rendering them ineffective".
Correct Answer:
B
Answer Choice Analysis:
(A) Maybe. It sounds like a "no cause, no effect" type answer. Ultimately, this doesn't lean in either way to whether the antibiotic resistance or the heavy-metal resistance comes first. We want to think that heavy-metal resistance comes first, if we're calling it the cause of the eventual antibiotic resistance.
(B) Yes, this looks like a stronger version of the "no cause, no effect" answer. When not exposed to heavy metals, we generally don't see antibiotic resistance. This is a helpful way of making it seem more plausible that "exposure to heavy metals" makes a causal difference. It also indirectly plays a #1 role .. it rules out the idea that "maybe it's just some chemical, like ammonia, in the sludge that is causing these effects."
(C) This weakens by providing a Reverse Causality explanation.
(D) Irrelevant. We weren't saying that sewage sludge normally has antibiotics in it.
(E) This seems to weaken a tad by giving us a covariation mismatch. These bacteria are "no cause, effect". They're not exposed to the sewage sludge, but they still have the same observed traits.
Takeaway/Pattern: We're more likely to see #1 alternative explanations show up on Weaken / Flaw / Necessary Assumption. We're slightly more likely to see #2 plausibility answers show up on Strengthen. The most common type of answer that increases the plausibility of the author's causal story is a Covariation answer: evidence that cause/effect appear and disappear in tandem. When bacteria is in sludge but NOT exposed to heavy metals, it doesn't have these effects. That makes our scientific brain believe that "exposure to heavy metals" is a causal difference maker. This answer is essentially 'the control group'. If you want to figure out whether heavy metals make a difference, keep the rest of the scenario the same (still study bacteria in sewage sludge) and see if the observed traits of A and B are still present.
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